Phosphatidylcholine Homeostasis and Liver Failure

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Phosphatidylcholine homeostasis and liver failure.

In mammals, the only endogenous pathway for choline biosynthesis is the methylation of phosphatidylethanolamine to phosphatidylcholine (PC) by phosphatidylethanolamine N-methyltransferase (PEMT) coupled to PC degradation. Complete choline deprivation in mice by feeding Pemt(-/-) mice a choline-deficient (CD) diet decreases hepatic PC by 50% and is lethal within 5 days. PC secretion into bile is...

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Phosphatidylcholine and choline homeostasis.

Phosphatidylcholine (PC) is made in mammalian cells from choline via the CDP-choline pathway. Animals obtain choline primarily from the diet or from the conversion of phosphatidylethanolamine (PE) to PC followed by catabolism to choline. The main fate of choline is the synthesis of PC. In addition, choline is oxidized to betaine in kidney and liver and converted to acetylcholine in the nervous ...

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Phosphatidylcholine: A Superior Protectant Against Liver Damage

Phosphatidylcholine (PC) is one of the most important support nutrients for the liver. PC is a phospholipid, a large biological molecule that is a universal building block for cell membranes. A cell’s membranes are its essence: they regulate the vast majority of the activities that make up life. Most liver metabolism occurs on cell membranes, which occupy about 33,000 square meters in the human...

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Phosphatidylcholine metabolism in rat liver after partial

The effect of partial (70 %) hepatectomy on phosphatidylcholine (PC) synthesis in rat liver was investigated during the first 4 post-operative days. Between 4 and 96 h after partial hepatectomy, the mass of PC increased from 30% to 80% of sham-operation values, being comparable with the restoration of total liver mass after partial hepatectomy. Relative to control (sham-operation), the incorpor...

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Liver Failure and Liver Disease

Cirrhosis with ascites is associated with impaired renal function accompanied by sodium and water retention. Although it has been suggested thatmediators such as nitric oxide play a role in the development of renal failure in this situation, other mechanisms underlying the process are not well understood. This study examined the role of oxidative stress in mediating renal damage during the deve...

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ژورنال

عنوان ژورنال: Journal of Biological Chemistry

سال: 2005

ISSN: 0021-9258

DOI: 10.1074/jbc.m508575200